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. 2021 Aug 20;19:247. doi: 10.1186/s12957-021-02363-7

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — HOXA9 was a direct target of miR-638 in breast cancer. (A) HOXA9 had binding sites with miR-638. (B) Luciferase reporter assay. (C) miR-638 had negative correlation with HOXA9. (D, E) The expression of HOXA9 were observed in MCF-7 cells containing miR-638 mimics or inhibitor **P <0.01