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. 2021 Aug 17;14:4001–4017. doi: 10.2147/JIR.S324232

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© 2021 Wang et al.

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The role of ADA2AR on VECs viability and proliferation. (A) The box plots showed the CCK-8 assay results. ZM241385 suppressed the VECs viability but CGS21680 could not enhance cell viability in bone fracture environment in vitro. * p<0.05 compared with DMSO treatment. n=5 for each group. (B) The results of EdU and PI assay. CGS21680 could not enhance the FITC and PE expression in cells while ZM decreased the FITC and PE expression. (C) Half-box graphs analyzed the results of EdU and PI assay, *p<0.05. (D) Cell scratch assay results. CGS21680 could not promote wound closure and ZM 241385 inhibited wound closure at 24, 48h. Images ×40; original scale bar, 100 μm. (E) Half-box graphs showed the difference between groups, *p<0.05. n=5 for each group.