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The mechanisms underlying SARS-CoV-2 induced ARDS [1] a hypothetical model of SARCoV-2 virus demonstrating infectious pathways to cell entery, induce inflammation, ARDS and procoagulation state. SARS CoV-2; Severe Acute Respiratory Syndrome Coronavirus-2, IL-6; Interleukin −6, TNFα; Tumor necrosis alpha, IL 1-β; Interleukin 1-beta, ACE II; Angiotensin converting enzyme II, Ang II; Angiotensin II (Created with BioRender.com).
