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. 2021 Jul 15;41(9):2454–2468. doi: 10.1161/ATVBAHA.121.316539

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© 2021 The Authors.

Arteriosclerosis, Thrombosis, and Vascular Biology is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 6. — A model of myocardial infarction (MI) induces dynamic changes in cardiovascular extracellular vesicles (EVs) from adult zebrafish. A–C, Quantification of the number of calcein+ (A), myl7(mCherry)+ (B), and kdrl(mCherry)+ EVs in uninjured and injured hearts at 24 hpi. D, Histogram of DLS analysis reveals a significant shift in the size of overall EVs at 24 hpi compared with uninjured hearts. Statistical analysis in A–C, 2-tailed Mann-Whitney U tests. Statistical analysis in D: a custom permutation test using total variation distance was used to test the null hypothesis that control and 24 hpi distributions were the same.