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. 2021 Aug 8;22(4):1139. doi: 10.3892/etm.2021.10573

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Copyright: © Huang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Overexpression of CTRP9 reduces H/R-induced damage in NRCMs. (A) Transfection with Ad-CTRP9 could markedly increase cell viability. (B) LDH, (C) CK and (D) CK-MB levels were markedly reduced following CTRP9 overexpression after H/R. Data are presented as the mean ± SD (n=6). ^*P<0.05 vs. the control group. ^#P<0.05 vs. H/R or Ad-GFP. CTRP9, C1q/TNF-related protein 9; Ad-CTRP9, adenovirus encoding CTRP9; H/R, hypoxia/reoxygenation; NRCM, neonatal rat cardiomyocyte; LDH, lactate dehydrogenase; CK, creatine kinase; CK-MB, CK-myocardial band.