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. 2021 Aug 17;9(8):1029. doi: 10.3390/biomedicines9081029

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© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Venography. BPC 157 medication (a). After ligation of superior mesenteric vein, clear visualization of collaterals bypassing defect of superior mesenteric vein. Venography clearly showed the superior mesenteric vein–inferior anterior pancreaticoduodenal vein–superior anterior pancreaticoduodenal vein–pyloric vein–portal vein pathway, reestablished superior mesenteric vein and portal vein connection and reestablished blood flow, the mesenterico-portal confluent and bypassing defect of superior mesenteric vein. Contrast flow into main portal trunk, portal bifurcation on the left and right portal vein and intraparenchymal portal branches on the porto-venous (or parenchymal) liver phase. Without medication, control rats (b) with the ligated superior mesenteric vein regularly show poor presentation in the venography (1 mL through 30 sec in the superior mesenteric vein). Commonly, they respond with the rapid rupture of the superior mesenteric vein and lack of the activated collaterals.