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Contribution of the immune system cells and inflammation in the pulmonary arterial hypertension. Here, initiating or predisposing factors lead to immune system stimulation, which results in endothelial and vascular cells proliferation, autoantibodies formation, T helper cells (Th) and T regulatory cells (Treg) proliferation, as well as reduction of T cytotoxic cells (Tc). All these phenomena affect pulmonary circulation and result in heart damage.
