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. 2021 Jul 30;9(8):1632. doi: 10.3390/microorganisms9081632

Figure 1.

Figure 1

The etiopathogenesis of thrombotic event caused by the S proteins released by the Sars-CoV-2 virus. The current hypothesis is substantially based on three momenta: (i) The subversive activity perpetrated by the virus at the very beginning of the initial differentiation phase of CD34+ HSCs towards the myeloid lineage. (ii) The presence of SNPs at various level of the immune system’s both inflammatory and modulatory response. (iii) SNPs present of gene controlling the coagulation system (MTHFR and Leiden).