Table 1 –
Cohorts A + B | Cohort A a | Cohort B b | |
---|---|---|---|
Olaparib arm (n) | 138 | 84 | 54 * |
With radiographic response, n (%) | 30 (22) | 28 (33) | 2 (4) * |
Control arm (n) | 67 | 43 | 24 * |
With radiographic response, n (%) | 3 (4) | 1 (2) | 2 (8) * |
Response to olaparib vs control | |||
Adjusted OR (95% CI)c | 5.93 (2.01–25.4) | 20.9 (4.18–379) | |
Unadjusted OR (95% CI)c | 5.93 (1.72–31.3) * | 21.0 (3.15–879)* | 0.42 (0.03–6.25) * |
RR (95% CI)c | 4.86 (1.54–15.3)* | 14.3 (2.02–101) * | 0.44 (0.07–2.97) * |
RD in nercentage noints (95% CI) d | 17 (9–26) * | 31 (20–42) * | −5 (−17 to 8) * |
CI = confidence interval; OR = odds ratio; RD = risk difference; RR = risk ratio.
Not reported by de Bono et al. (2020) [2] but calculable from the data provided.
Patients with tumor DNA alterations in the three main homologous recombination repair genes (BRCA2, BRCA1, or ATM).
Patients with tumor DNA alterations in 12 additional genes (BRIP1, BARD1, CDK12, CHEK1, CHEK2, FANCL, PALB2, PPP2R2A, RAD51B, RAD51C, RAD51D, or RAD54L).
OR and RR values >1 favor olaparib. ORs in the main article were adjusted for stratification factors; however, differences between adjusted and unadjusted point estimates are negligible. RRs are also presented here because ORs always exaggerate results when an outcome is common [8]: compare the OR of 21.0 to the RR of 14.3 for cohort A.
RD values >0 percentage points favor olaparib.