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. 2021 Aug 20;24(4):738. doi: 10.3892/mmr.2021.12378

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Copyright: © Yuan et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Interference with CXCR3 inhibits HBV-LX-2 cell viability and migration and promotes cell apoptosis. (A) Interference with CXCR3 inhibits HBV-LX-2 cell viability. (B) Interference with CXCR3 inhibits the migratory rate of HBV-LX-2 cells. (C) Representative images of HBV-LX-2 cell migration following transfection with siCXCR3. Magnification, ×100. (D) Interference with CXCR3 promotes HBV-LX-2 cell apoptosis. (E) Representative images of HBV-LX-2 cell apoptosis following transfection with siCXCR3. *P<0.05 vs. Control; ^#P<0.05 vs. NC. CXCR3, C-X-C motif chemokine receptor 3; HBV, hepatitis B virus; siRNA/si, small interfering RNA; NC, siRNA-negative control.