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. 2021 Aug 20;24(4):738. doi: 10.3892/mmr.2021.12378

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Copyright: © Yuan et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Effect of siCXCR3 on the expression levels of COL1A1 and the proteins in the TLRs/MyD88 pathway in HBV-LX-2 cells. (A) Interference with CXCR3 increases the expression of IL-1β in HBV-LX-2 cells, as determined by ELISA. (B) Interference with CXCR3 inhibits the protein levels of TLR4, MyD88, COL1A1 and BIRC5 in HBV-LX-2 cells, as determined via western blotting. *P<0.05 vs. Control; ^#P<0.05 vs. NC. CXCR3, C-X-C motif chemokine receptor 3; siRNA/si, small interfering RNA; NC, siRNA-negative control; TLR, Toll-like receptor; MyD88, myeloid differentiation primary response protein 88; COL1A1, collagen type I α 1 chain; BIRC5, baculoviral IAP repeat containing 5; NALP3, NLR family pyrin domain containing 3.