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. 2021 May 22;73(4):1063–1078. doi: 10.1007/s43440-021-00269-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — The role of the CX3CL1–CX3CR1 signaling pathway in the pathology of schizophrenia. In physiological conditions, the interaction of CX3CL1 with CX3CR1 is essential for the regulation of multiple processes in the brain. The disturbances within this axis and subsequent disruptions within these mechanisms implicate the CX3CL1–CX3CR1 dyad in schizophrenia