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. 2021 Aug 3;24(5):828–837. doi: 10.1007/s11102-021-01178-9

Table 2.

Demographic, pathological, clinical, hormonal, imaging and therapeutic characteristics of the 43 reported cases of PM from NENs

Total number of cases n = 43 (%)
Sex n = 43 (%)
 M 19 (44)
 F 24 (56)
Median age (y) 58
Primary NEN histology n = 43 (%)
 SCLC 21 (49)
 Atypical lung carcinoid 6 (14)
 Pancreatic NEN 4 (9)
 MTC 3 (7)
 Thymic carcinoid 2 (5)
 Unknown primary NEN 2 (5)
 Breast NEN 2 (5)
 Ileal NEN 1 (2)
 Merkel cell carcinoma 1 (2)
 Lung LCNEC 1 (2)
PM as first manifestations of NEN 23 (53)
Clinical presentation n = 41 (%)
 Visual field deficit 27 (66)
 Headache 20 (49)
 Constitutional symptoms (asthenia, weight loss) 18 (44)
 Other ocular symptoms (diplopia, blurred vision) 18 (44)
 Ocular palsy 9 (22)
 Other neurological symptoms (gait disturbance, confusion, memory loss) 6 (15)
Hormonal alterations n = 37 (%)
 Hypopituitarism (deficit of at least one pituitary axis) 27 (73)
 Hyperprolactinemia 21 (57)
 Gonadotropins deficit 19 (51)
 ACTH deficit 15 (40)
 TSH deficit 15 (40)
 DI 13 (35)
 Panhypopituitarism (complete anterior pituitary deficit + DI) 6 (16)
Imaging characteristics n = 36 (%)
 Suprasellar extension 30 (83)
 Compression/displacement of pituitary stalk 20 (56)
 Bone erosion 9 (25)
 Invasion of cavernous sinus 7 (19)
 Involvement of other brain structures 7 (19)
 Thickening/involvement of pituitary stalk 4 (11)
Therapy n = 43 (%)
 Surgery 32 (74)
 Radiotherapy 22 (51)
 Chemotherapy 19 (44)
 PRRT 2 (5)
Median survival [months, (95% CI)] 14 (9–21)

PM pituitary metastases, NEN neuroendocrine neoplasm, SCLC small cell lung cancer, MTC medullary thyroid cancer, LCNEC large cell neuroendocrine carcinoma, DI diabetes insipidus, PRRT peptide receptor radionuclide therapy, IQR interquartile range