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. 2021 Sep 4;11:170. doi: 10.1186/s13578-021-00682-6

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — ATG5 knockdown suppresses the inhibitory effects of PS-T. a MDA-MB-231-siNC cells and MDA-MB-231-siATG5 cells are treated with or without PS-T (5 μg/mL) for 24 h. The levels of each EMT marker in MDA-MB-231cells are quantified using NIH ImageJ software. (mean ± SD, *P < 0.05 and **P < 0.01). b The invasiveness of MDA-MB-231-siNC cells and MDA-MB-231-siATG5 cells treated with PS-T at 5 μg/mL is evaluated using Transwell invasion assays at 24 h (mean ± SD, **P < 0.01). c, d Migratory ability of MDA-MB-231-siNC and MDA-MB-231-siATG5 cells treated with PS-T at 5 μg/mL is evaluated using Transwell migration assays (c) and scratch assay (d) at 24 h (mean ± SD, *P < 0.05, **P < 0.01, and ***P < 0.001)