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. 2021 Aug;9(16):1300. doi: 10.21037/atm-21-3746

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2021 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Both A-CM and U-CM down-regulated the expression of anti-apoptosis proteins (BCL2 and MCL1) in MCF-7 and MDA-MB-231 cells. (A) Expression level of MCL1 and BCL2 protein in MCF-7 cells, after co-culture with A-CM or U-CM for 48 h. (B,C) Expression level of BCL2 and MCL1 protein in MDA-MB-231 cells, after co-culture with A-CM or U-CM for 48 h. (* indicates P<0.05; NC: means natural control group, which were treated with free DMEM/F12). ADSCs, adipose-derived stem cells; UMSCs, umbilical mesenchymal stem cells; A-CM, ADSCs-related medium; U-CM, UMSCs-related medium; DMEM, Dulbecco’s modified Eagle’s medium.