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. 2021 Feb 15;73(5):832–841. doi: 10.1093/cid/ciab139

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© The Author(s) 2021. Published by Oxford University Press for the Infectious Diseases Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs licence (https://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial reproduction and distribution of the work, in any medium, provided the original work is not altered or transformed in any way, and that the work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 5. — (A) Graph demonstrating the differentiation of analyte responses by principal component analysis loadings. Volunteers with higher principal component (PC) 1 loadings had significantly higher plasma concentration in a majority of the analytes compared to those with higher PC2 loadings (Wilcoxon rank sum test P < .05 [*], P < .01 [**], and P < .001 [***], N = 55); (B) A schematic illustration of the differentiation of analyte responses into volunteers with and without a cytokine storm; and Wilcoxon signed rank test (P < .05 [*], P < .01 [**], P < .005 [***], and P < .0005 [****]; paired/matched signed rank test in green, N = 31). Abbreviations: hbFGF, basic fibroblast growth factor; Flt, FMS-like tyrosine kinase; GM-CSF, granulocyte-macrophage colony-stimulating factor; hAHI, hyperacute HIV-1 infection; HIV-1, human immunodeficiency virus type 1; IFN, interferon; IL, interleukin; IP, interferon gamma-induced protein; MCP, monocyte chemoattractant protein; MDC, macrophage-derived chemokine; MIP, macrophage inflammatory protein; PlGF, placental growth factor; TARC, thymus and activation-related chemokine; TNF, tumor necrosis factor; VEGF, vascular endothelial growth factor.