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. 2021 Sep 7;18:81. doi: 10.1186/s12986-021-00601-z

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — The effects of SZP on the secretion of inflammatory cytokines and proteins in DN mice. The expression level of TNF-α (a). The expression level of MCP-1 (b). The expression level of IL-6 (c). The values presented are the means ± SD of six independent experiments (n = 6) and differences between mean values were assessed by ANOVA. NF-κB protein expressions in DN mice after SZP treatment (d). The values presented are the means ± SD of three independent experiments (n = 3) and differences between mean values were assessed by ANOVA. ###P < 0.01 versus sham control group, ***P < 0.01 versus DN group and *P < 0.05 versus DN group. TNF-α, Tumor necrosis factor-α; MCP-1, Monocyte chemotactic protein-1; IL-6, Interleukin 6. NF-κB, Nuclear factor-kappa B