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. 2021 Aug 26;9:666422. doi: 10.3389/fcell.2021.666422

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Copyright © 2021 Xie, Yao, Hu, Cai, Zhao, Yuan, Wu and Tang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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High-mobility group A1 (HMGA1) was upregulated during cardiac fibrosis. (A) The protein level of HMGA1 in mouse hearts after isoproterenol (ISO) insult (n = 6). (B) Immunohistochemical staining of HMGA1 in mouse hearts after ISO insult (n = 5). (C) The protein level of HMGA1 in fibroblasts after transforming growth factor-β1 (TGFβ1) insult (n = 6). (D) Immunofluorescence staining of fibroblasts after TGFβ1 insult (n = 5). *p < 0.05.