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. 2021 Sep 9;7:79. doi: 10.1038/s41531-021-00221-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a GAD disease enrichment analyses of TWAS genes. b KEGG pathway enrichment analyses of TWAS genes. c–e Top ten GO terms enriched in TWAS genes, including biological processes (c), cellular component (d), and molecular function (e). f GWAS Catalog enrichment analyses of TWAS genes. The histogram shows the expected number of genes with P < 0.01 based on 10,000 random permutations. The large red point shows the observed number of previously known PD genes that fall below this threshold.