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. 2021 Sep 9;11:17971. doi: 10.1038/s41598-021-97474-2

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Effects of QTN on FCA-induced alterations in protein expression of NF-kβ p65 (A), p-Ikβα (B), COX-2 (C), and P2X7 (D) in synovial tissues. Data are expressed as mean ± SEM (n = 4) and analyzed by one-way ANOVA followed by Tukey’s multiple range test. For comparison with AIA-control group: *p < 0.05, comparison with normal group: ^#p < 0.05 and comparison with one another: ^$p < 0.05. Normal group protein expression (lane 1); AIA control group protein expression (lane 2); Leflunomide group protein expression (lane 3) and QTN (5, 10, and 20 mg/kg) group protein expression (lane 4–6). AIA adjuvant-induced arthritis, LF leflunomide, QTN 3,5,7,3′,4′-pentahydroxy flavone, NF-kβ nuclear factor kappa beta, Ikβα nuclear factor of kappa light polypeptide gene enhancer in B cells inhibitor-α, COX-2 cyclooxygenase-2, P2X7 ATP-activated P2 purinergic receptors.