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. 2021 Sep 9;4:1052. doi: 10.1038/s42003-021-02597-x

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Box plots indicate the difference in HRD score (y-axis) across WGD and non-WGD tumors (x-axis). b Box plots indicate the difference in HRD score (y-axis) across subtypes by WGD status (x-axis). One-tailed Mann–Whitney U tests were performed to calculate the p-values for the comparison of medians in a and b. c Percentage of cases with HRD phenotype, defined as high HRD score (≥42) in tumors, across breast cancer subtypes (x-axis) stratified by WGD status. Overall, 64 HR + /HER2- (hormone receptor-positive/human epidermal growth factor receptor-negative), 13 HR + /HER2 + , 15 HER2 + , and 14 TNBC (triple-negative breast cancer) samples were used to perform analyses in a–c.