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. 2021 Aug 27;12:693701. doi: 10.3389/fphar.2021.693701

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Copyright © 2021 Wei, Hou, Liang, Fang, Dou, Wang, Sai, Xu, Ma, Zhang, Cheng, Wang and Wang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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SNS reduced liver pro-inflammatory cytokines, advanced glycation end-products (AGEs), and its receptor. (A) Serum levels of tumor necrosis factor alpha (TNFα). (B) Serum levels of interleukin 6 (IL6). (C) Serum levels of interleukin 1β (IL1β). (D) Serum levels of AGEs. (E) Serum levels of receptor for advanced glycation end-products (RAGE). (F) Liver levels of TNFα. (G) Liver levels of IL6. (H) Liver levels of IL1β. (I) Liver levels of AGEs. (J) Liver levels of receptor for RAGE. Data are shown as mean ± SD (n = 6). ^# p < 0.05, ^## p < 0.01, ^### p < 0.001 when compared with the Chow group, *p < 0.05, **p < 0.01, ***p < 0.001 when compared with the HFD group.