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. 2021 Aug 30;22(17):9421. doi: 10.3390/ijms22179421

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© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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1,8-cineol downregulates LPS-induced inflammation by blocking both NF-κB translocation and JNK pathway activation (Modified from Greiner et al. 2013 [68]). 1,8-cineol prevents the separation of IκB and NF-κBp65/p55 and then blocks NF-κB translocation from the cytoplasm to the nucleus, thus acting as a transcription factor for the expression of inflammation-related cytokines such as TNF-α and IL-8. : to inhibit the follow action.

Inline graphic — 1,8-cineol downregulates LPS-induced inflammation by blocking both NF-κB translocation and JNK pathway activation (Modified from Greiner et al. 2013 [68]). 1,8-cineol prevents the separation of IκB and NF-κBp65/p55 and then blocks NF-κB translocation from the cytoplasm to the nucleus, thus acting as a transcription factor for the expression of inflammation-related cytokines such as TNF-α and IL-8. : to inhibit the follow action.