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. 2021 Sep 7;32(15):1255–1262. doi: 10.1097/WNR.0000000000001719

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Copyright © 2021 The Author(s). Published by Wolters Kluwer Health, Inc.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Fig. 2 — The neurological and cognitive function of TBI rats was improved by CCR2 antagonist (RS504393) or p38 inhibitor (SB203580). (a) The mNSS score of TBI rats decreased after the application of high-dose CCR2 antagonists. (b) The mNSS score of TBI rats decreased after the application of high-dose p38 inhibitor. (c) The escape latency of TBI rats was reduced after the application of a high-dose CCR2 antagonist or p38 inhibitor. (d) The number of platform crossings of TBI rats increased after the application of high-dose CCR2 antagonist or p38 inhibitor. Values are expressed as mean ± SEM (n = 8/group). ^***P < 0.001, ^*P < 0.05 vs. TBI + vehicle group. ^###P < 0.001, ^##P < 0.01, vs. sham group. TBI, traumatic brain injury; mNSS, modified neurological severity score.