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. 2021 Sep 7;24(5):780. doi: 10.3892/mmr.2021.12420

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Copyright: © Lv et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 7. — Overexpressed ATF2 reverses the suppression of malignant properties in HCC cells caused by ZSCAN16-AS1 depletion. (A) ATF2 was overexpressed in HCC cells following transfection with pcDNA3.1-ATF2, which was corroborated by western blotting. ZSCAN16-AS1-depleted HCC cells were further transfected with pcDNA3.1-ATF2 or pcDNA3.1. Cellular (B) proliferation, (C) apoptosis, (D) migration and (E) invasion (magnification, ×200) were investigated. **P<0.01. ATF2, activating transcription factor 2; HCC, hepatocellular carcinoma.