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. 2021 Sep 13;4:1067. doi: 10.1038/s42003-021-02607-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Mutation profiles in 26 colorectal tumoroids and 224 TCGA colorectal cancers. Counts of SNVs and InDels, and proportions of nucleotide transitions and transversions are reported, split into distinct hypermutated and nonhypermutated cases. b Mutation frequencies of major colorectal cancer driver genes for tumoroids and TCGA-analyzed cancers. c Proportions of samples with relative DNA copy-number alterations for tumoroids and TCGA-analyzed cancers. d Genome-wide DNA copy-number aberrations for tumoroids stratified into nonhypermutated and hypermutated (MSI-H) cases.