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. 2021 Sep 14;11(9):e561. doi: 10.1002/ctm2.561

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© 2021 The Authors. Clinical and Translational Medicine published by John Wiley & Sons Australia, Ltd on behalf of Shanghai Institute of Clinical Bioinformatics

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Predicted drug resistance and target genes in GC. (A) Prediction of drug and drug target genes using GDSC. (B) Protein‐protein interactions among predicted candidate genes related with ALT‐like in GC (hub gene: NR3C1). (C) Schematic diagram of regulation in GC. (D) Kaplan–Meier plots showing overall survival rates for NR3C1‐high and ‐low groups. p‐Values were calculated using the log‐rank test. p = 0.0032 for NR3C1 expression in TCGA STAD. (E) Boxplot of NR3C1 expression in high‐ALT and low‐ALT‐like tumors in our cohort. Abbreviations: Alternative lengthening of telomeres (ALT); gastric cancer (GC); and telomere maintenance mechanism (TMM)