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. 2021 Sep 1;12:666430. doi: 10.3389/fneur.2021.666430

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Copyright © 2021 Wu, Kumal, Lu, Li, Mao, Tang, Nie, Liu, Sun, Liu and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Synapse formation in the cortex and hippocampus deteriorated in APP/PS1 mice after TBI. (A) Representative fluorescence images of cortical and hippocampal (CA1, CA3, and DG) tissues from in C57BL/6, C57BL/6 TBI, APP/PS1, and APP/PS1 TBI group mice immunostained for SYN (in red) and subjected to nuclear staining with DAPI (in blue). (B) The fluorescence intensity of SYN in the cortex and in the hippocampus (CA1, CA3, and DG). The error bars represent the SEM (n = 8/group). ns, non-significant; ^*p < 0.05; ^**p < 0.01; ^***p < 0.001.