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. 2021 Sep 9;24(5):789. doi: 10.3892/mmr.2021.12429

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — INHBA promotes lung cancer cell invasion by negatively regulating Hippo signaling. Rescue experiments showed that INHBA overexpression-induced promotion of NSCLC (A) invasion and (B) migration were reversed following VP treatment in H1299 cells. Original magnification, ×200. (C) Rescue experiments showed that INHBA overexpression-induced increases in N-cadherin, vimentin and Snail expression levels, and decreases in E-cadherin, pYAP expression levels and the pYAP/YAP ratio were also reversed after VP treatment in H1299 cells. *P<0.05, **P<0.01, ***P<0.001. INHBA, inhibin βA; YAP, yes-associated protein; p, phosphorylated; VP, Verteporfin.