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. 2021 Sep 6;24(5):775. doi: 10.3892/mmr.2021.12415

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Copyright: © Lin et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — PHI treatment suppresses the inflammatory response in LPS-induced BEAS-2B cells. (A) The chemical structure of PHI. (B) The effects of PHI on the viability of BEAS-2B cells were detected via an MTT assay. *P<0.05 vs. control. (C) The effects of PHI on the viability of BEAS-2B cells induced by LPS were detected via an MTT assay. ***P<0.001 vs. control; ^##P<0.01 vs. LPS group. (D-F) The expression levels of inflammatory factors were measured by ELISA. ***P<0.001 vs. control; ^#P<0.05, ^###P<0.001 vs. LPS group. (G) The levels of inflammation-associated proteins were detected via western blotting. ***P<0.001 vs. control; ^#P<0.05, ^##P<0.01, ^###P<0.001 vs. LPS group. PHI, phillygenin; LPS, lipopolysaccharide; cox-2, cyclooxygenase-2; iNOS, inducible nitric oxide synthase.