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. 2021 Sep 1;11:641453. doi: 10.3389/fonc.2021.641453

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Copyright © 2021 Cao, Min, Liu, Tian, Jiang, Liu, Shao, Cheng, Zhu, Guo and Li

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Cav 1-induced inhibition of apoptosis of gastric cancer cells and association between Rictor and Cav 1. (A) Transfected Cav 1 plasmid could inhibit the apoptosis of SGC-7901 cells (n=9). (B) Transfected Cav 1 plasmid could inhibit the apoptosis of AGS cells (n=9). Values represent the Means ± SD, *P < 0.05 and ***P < 0.001 were calculated using Student’s t-test. (C) Western blot analysis of the increase of Cav 1 expression with Rictor overexpression in SGC-7901 cells. (D) Western blot analysis of the increase of Cav 1 expression with Rictor overexpression in AGS cells. (E, F) Immunofluorescence showed no co-localization between Rictor and Cav 1. (G) Western blot analysis of p-Akt and Cav 1 levels increased after transfection of SGC-7901 cells with Rictor plasmid. (H) Western blot analysis of p-Akt and Cav 1 levels increased after transfection of AGS cells with Rictor plasmid. β-actin served as a loading control.