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. 2020 May 8;121(5):1207–1215. doi: 10.1007/s13760-020-01359-6

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Effects of sevoflurane on autophagy and autophagic flux of APP/PS1 mice. a and b Transmission electron microscope was employed to analyze the number of autophagosomes. And the data displayed that there was a remarkable upregulation of autophagosomes at the distal axon after treatment of sevoflurane (n = 4 per group). c and d Furthermore, western blot analysis showed sevoflurane could not only enhance the levels of LC3II but also increase the expression of P62 (n = 5 per group). (*P < 0.01, vs. Control group; ^#P > 0.05, vs. Control group)