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. 2020 Aug 19;11(8):e00197. doi: 10.14309/ctg.0000000000000197

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© 2020 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of The American College of Gastroenterology

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Figure 1. — IL-22BP is present in the liver and produced in the gut, and plasma levels are markedly reduced. (a) Immunohistochemical staining for IL-22BP in paraffin-embedded sections of liver from patients with alcoholic hepatitis and healthy liver and colon. Representative sections displayed. (b) Plasma IL-22BP by ELISA from the portal vein and peripheral blood in a cohort of patients with cirrhosis and in (c–e) peripheral plasma from patients with alcoholic hepatitis (AH) and controls; nonalcoholic fatty liver (NAFL) and steatohepatitis (NASH) and stable alcoholic (Alc) cirrhosis (analysis of variance, T-test, *P < 0.05,**P < 0.01, ***P < 0.001, bars represent means and SDs).