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. 2021 Sep 15;41(37):7831–7847. doi: 10.1523/JNEUROSCI.1037-20.2021

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Copyright © 2021 Pommer et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license, which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Figure 6. — CP-93129 changes average IPSC amplitude and synaptic release probability in local axon collaterals. A, Statistical analysis of average IPSC amplitude change in D2-SPNs (D1-Cre mice) during baseline, drug treatment, and washout (N = 12). B, Comparison of average IPSC amplitude of drug treatment in D2-SPNs (D1-Cre mice, N = 12) with control group (N = 6). Mean ± SD. C, Analysis of average group synaptic success rate for D1→D2-SPN connections (D1-Cre mice, N = 12). D, Statistical analysis of average IPSC amplitude change in D1-SPNs (A2a-Cre mice) during baseline, drug treatment, and washout (N = 6). E, Comparison of average IPSC amplitude of drug treatment in D1-SPNs (A2a-Cre mice, N = 6) with control group (N = 6). Mean ± SD. F, Analysis of average group synaptic success rate for D2→D1-SPN connections (A2a-Cre mice, N = 6). ns, p > 0.05; *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001.