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Influence of emodin on PPARα/γ-AMPK, fatty acid oxidation, and fatty acid biosynthesis pathways after IAV infection in vivo. Mice were treated as mentioned in Figure 8. On day 6 p.i., lung tissues were collected and homogenated. The mRNA levels of target genes were measured by qRT-PCR (a, b). The phosphorylation levels of AMPK and ACC were determined by an ELISA assay (c). Data shown were the mean ± SD. n = 6. ∗P < 0.05 vs. the only IAV-infected control.
