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. 2021 Sep 16;18:211. doi: 10.1186/s12974-021-02255-3

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Changes of sEH expression in the perithalamic lesion site of CPSP rats. a Representative Western blot bands were presented on the left, with data analysis shown on the right revealing that sEH protein expression is increased around CPSP rat thalamic lesion site, beginning at postlesion day 7 and at least lasting to day 28 after lesion, compared with the sham control. Values are expressed as mean ± SD. GAPDH serves as loading control. **P < 0.01, ***P < 0.001, n = 5 rats per group, one-way ANOVA followed by Bonferroni post hoc test. b The tissue sections from the injured side thalamus are double immunostained with sEH (red) and reactive astrocyte marker GFAP (green), microglial marker IBA1 (green), or neuron-specific nuclei marker NeuN (green). c The percentage of different cell types in sEH-positive cells on day 14 after CPSP induction. n = 4 rats per group, *P < 0.05, **P < 0.01, compared with the sham group. Scale bars = 500 μm. IBA1, ionized calcium-binding adapter molecule 1; NeuN, neuronal nuclei; GFAP, glial fibrillary acidic protein