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. 2021 Aug 19;2(5):zqab041. doi: 10.1093/function/zqab041

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© The Author(s) 2021. Published by Oxford University Press on behalf of American Physiological Society.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — LTCC activity in FA and chronic SHS-exposed arterial myocytes. (A) Representative consecutive LTCC single-channel traces recorded from FA and 12 wk SHS-exposed arterial myocytes and (B) corresponding ensemble average currents. (C) Magnified region from the dotted squares in panel A showing coupled channel events. (D–H) Scatter plot of the mean ± SEM for nP_o (D), P_o (E), # channels (F), coupled frequency (G), and coupled strength (H) of LTCC single-channel data from FA (n = 16 cells) and 12 wk SHS-exposed (n = 21 cells) arterial myocytes. *P < .05 compared to FA, Mann–Whitney. (I) Representative consecutive LTCC single-channel traces in presence of the LTCC channel blocker nifedipine (1 μM).