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. 2021 Sep 17;21:501. doi: 10.1186/s12935-021-02209-y

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — CRC patient survival is associated with LBX2-AS1 expression levels. A Kaplan–Meier analysis using the online tool GEPIA indicated that patients whose CRC tissues had high LBX2-AS1 expression exhibited significantly reduced overall survival relative to LBX2-AS1-low expressing cases. B Analysis of disease-free survival in the dataset from A showed no significant differences between high and low LBX2-AS1 expressing cases. C Kaplan–Meier analysis of CRC patients from cohort A showing high LBX2-AS1 CRC expression cases exhibited significantly reduced overall survival relative to LBX2-AS1-low expression cases (P = 0.003). D Analysis of disease-free survival in the dataset from C showed no significant differences between high and low LBX2-AS1 expressing cases. For A–D, log rank test