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. 2020 Dec 12;70(10):1872–1883. doi: 10.1136/gutjnl-2020-322468

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© Author(s) (or their employer(s)) 2021. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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The protective effects of bacterial-derived OGAs BtGH84 and AkkGH84 against TNBS-induced or OXA-induced colitis. (A, F) Schematic outline of the TNBS-colitis model (A), OXA-colitis model (F) and their treatment approach. (B–E, G–J) Disease severity of colitis mice: body weight change (B, G), colon length (C, H); histology manifestation on H&E staining (D, I), scale bar: 200 µm and inflammation score (E, J). Data represent the mean of three repeats per group with the SD; *P<0.05, **P<0.01, ***P<0.001. OGA, O-GlcNAcase’ OXA, oxazolone; TNBS, 2,4,6-trinitrobenzenesulfonic acid.