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. 2021 Sep 9;8:694238. doi: 10.3389/fcvm.2021.694238

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Copyright © 2021 Liang, Cao, He, Li, Huang, Ma, Li, Huang, Huang and Hu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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HOXB4 served as a transcription factor for AKR1C3. (A) Prediction of the potential transcription factor of AKR1C3. The size of the circle represents the predicted possibility. (B) AKR1C3 was significantly correlated with HOXB4 and NKX6-1. (C) NKX6-1 was upregulated in acute myocardial infarction samples. (D,E) The expression values of AKR1C3 and HOXB4 were tightly associated in the GSE48060 (D) and Shunde (E) cohorts. (F) The results of the luciferase reporter assay. ****p < 0.0001; ns, not significant.