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. 2021 Sep 24;41(10):e00115-21. doi: 10.1128/MCB.00115-21

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FIG 2 — miR-205-5p was the target of HNRNPU-AS1. (A) miR-205-5p binding sites to HNRNPU-AS1 3′-UTR. (B) Binding relation of miR-205-5p with HNRNPU-AS1 verified through dual-luciferase reporter assay. (C) The dramatic enrichment of HNRNPU-AS1 was detected in biotinylated miR-205-5p by RNA pulldown assay. (D) RT-qPCR assay on miR-205-5p expression in the HNRNPU-AS1 inhibition or overexpression group. (E) miR-205-5p levels in CC cells measured through RT-qPCR. (F) miR-205-5p levels in CC and matched noncarcinoma samples (n = 41) detected by RT-qPCR. (G) Spearman correlation analysis regarding HNRNPU-AS1 with miR-205-5p levels. (H) KM analysis on OS for CC cases. (I) KM survival analysis on OS for HNRNPU-AS1 combined with miR-205-5p in CC patients. Data are displayed in the form of means ± SD from 3 individual assays. **, P < 0.01 versus siNC and vector groups.