Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2021 Mar 17;33:253–264. doi: 10.1016/j.jare.2021.03.005

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2021 The Authors. Published by Elsevier B.V. on behalf of Cairo University. This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

PMC Copyright notice

Fig. 4 — TMEM16A overexpression promoted migration and invasion of T47D cells via the activation of EGFR/STAT3/ROCK1 signaling. A, B. Wound healing assay (A) and transwell assay (B) showed the migration (A) or invasion (B) of T47D cells treated with empty vector or T16A-OE plasmids in the absence or presence of gefitinib (1 μM) or JSI-124 (0.1 μM). n = 3. *p < 0.05 vs vector; #p < 0.05 vs T16A-OE. C. Wound healing assay showed the migration of T47D cells treated with empty vector or T16A-OE plasmids in the absence or presence of Y-27632 (10 μM). n = 3. *p < 0.05 vs vector; #p < 0.05 vs T16A-OE. D, E. Transwell assay showed the migration (D) and invasion (E) of T47D cells treated with empty vector or T16A-OE plasmids in the absence or presence of Y-27632 (10 μM). n = 3. *p < 0.05 vs vector; #p < 0.05 vs T16A-OE.