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. 2021 Mar 17;33:253–264. doi: 10.1016/j.jare.2021.03.005

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© 2021 The Authors. Published by Elsevier B.V. on behalf of Cairo University. This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 5 — Inhibition of TMEM16A channel activity reduced the migration and invasion of T47D cells. A, B. Wound healing assay (A) and transwell assay (B) showed the migration (A) and invasion (B) of T47D cells treated with empty vector, T16A-OE plasmids, and T16A-OE + T16Ainh-A01 (20 μM). n = 3. *p < 0.05 vs vector; #p < 0.05 vs T16A-OE. C. ROCK1 protein expression in T47D cells transfected with empty vector or plasmids containing WT-TMEM16A/Δ₄₄₄EEEEEAVKD₄₅₂-TMEM16A (Δ444-452) mutants. n = 3. *p < 0.05 vs vector, #p < 0.05 vs WT-TMEM16A. D, E. Wound healing assay (D) and transwell assay (E) showed the migration (D) and invasion (E) of T47D cells treated with empty vector or plasmids containing WT TMEM16A or Δ444-452 mutants. n = 3. *p < 0.05 vs vector; #p < 0.05 vs WT-TMEM16A.