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SARS-CoV-2 infecting cells in lung tissue through the interaction with angiotensin-converting enzyme 2 (ACE2), promoting the generation of pro-inflammatory cytokines (cytokine storm). This process attracts “defense” cells such as macrophages, monocytes, and T cells to the site of infection, triggering the inflammation process. The result of this inflammation is lung tissue damage. Adapted from Tay et al., 2020 [102].
