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The opposite effects of reactive oxygen species (ROS) in neutrophil tumor biology. ROS produced by neutrophils might either provide the cytotoxic hit resulting in tumor cell death or induce proliferation of the tumor cells. In addition, ROS might lead to tissue injury, angiogenesis and mutagenesis, which further support the cancerous phenotype. On top of this, neutrophil-produced ROS can suppress cytotoxic T cell and NK cell functions necessary for the ultimate tumor cell rejection.
