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. 2021 Sep 4;162(12):bqab193. doi: 10.1210/endocr/bqab193

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© The Author(s) 2021. Published by Oxford University Press on behalf of the Endocrine Society.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs licence (https://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial reproduction and distribution of the work, in any medium, provided the original work is not altered or transformed in any way, and that the work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 6. — Persistent histone hyperacetylation and increased expression of Dpp-4 during and after dexamethasone treatment are reproducible in the cultured cells. (A) Schematic representation of the in vitro study of Dexa treatment. (B) Dpp-4 expression. (C) H3K9 acetylation level of the promoter region of Dpp-4. (D) HAT activity. (E) HAT gene expressions. (F) HDAC activity. (G) HDAC gene expressions. (H) Sirtuin activity. (I) Sirtuin gene expressions. Dexa, dexamethasone; EOMA, hemangioendothelioma; HAT, histone acetyltransferase; HDAC, histone deacetyltransferase. *P < 0.05, **P < 0.01 vs control. N = 8 per group.