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A putative schematic diagram of molecular mechanisms of CYP ω-hydroxylases-mediated eicosanoids on inflammation. * 19-HETE may take a pro-inflammatory role in chronic kidney disease. However, it may also act as an anti-inflammatory mediator since it antagonized 20-HETE-induced inflammation. In addition, 19(S)-HETE was reported to be more active than 19(R)-HETE against Ang II-cell hypertrophy.
