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. 2021 Sep 17;24(5):808. doi: 10.3892/mmr.2021.12448

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Copyright: © Xiang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, reproduction and adaptation in any medium and for any purpose provided that it is properly attributed. For attribution, the original author(s), title, publication source (PeerJ) and either DOI or URL of the article must be cited.

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Figure 3. — DMN, YGD and HGF regulate the expression of α-SMA, CXCR4, ERK1/2, NF-κB p65 and β-catenin. Mice were injected with DMN or normal saline for 4 weeks. Following 4 weeks of DMN treatment, mice were treated with YGD or HGF for a further 4 weeks. Liver sections were stained for (A) α-SMA (B) CXCR4. (C) ERK1/2.

Figure 3. — DMN, YGD and HGF regulate the expression of α-SMA, CXCR4, ERK1/2, NF-κB p65 and β-catenin. Mice were injected with DMN or normal saline for 4 weeks. Following 4 weeks of DMN treatment, mice were treated with YGD or HGF for a further 4 weeks. Liver sections were stained for (A) α-SMA (B) CXCR4. (C) ERK1/2.