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. 2021 Sep 16;11:738822. doi: 10.3389/fonc.2021.738822

Table 3.

Heterozygous FLCN variants identified on clinically accredited diagnostic testing.

Gene and transcript Variant ACMG classification Exon/description
FLCN NM_144997.5 c.249+1_2delGCinsA Pathogenic Exon 5 (intronic) 125 bp deletion, canonical splice site
FLCN NM_144997.5 c.384C>G, p.Ser128Arg VUS Exon 5 missense
FLCN NM_144997.5 c.469_471delTTC, p. Phe157del Likely pathogenic Exon 6, in-frame deletion
FLCN NM_144997.5 c.763C>T, pHis255Tyr Likely pathogenic Exon 7, missense
FLCN NM_144997.5 c.1157C>G, p. Ser386* Pathogenic Exon 11, nonsense
FLCN NM_144997.5 c.1177-5_1177-3-del Pathogenic Exon 11, intronic deletion of 3 bp close to conserved splicing acceptor site of exon 11, frameshift cause premature stope codon
FLCN NM_144997.5 c.1285dupC, p. His249Profs*27 Pathogenic Exon 11, 1 bp duplication, frameshift (nonsense)
FLCN NM_144997.5 c.1318_1334dup, p.Leu449Glnfs*25 Pathogenic Exon12, 17 bp duplication, frameshift, nonsense
FLCN NM_144997.5 c.1333G>A, p.Ala445Thr VUS Exon 12, missense

*This is standard HGVS amino acid nomenclature for a nonsense substitution variant.