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. 2021 Aug 10;112(10):4176–4186. doi: 10.1111/cas.15089

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© 2021 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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POU4F2 is a tumor promotor in CRC cell lines. A, B , Wound healing ability of POU4F2‐knockdown HCT116 (A) and POU4F2‐knockdown RKO (B) cells. C, D, Migration and invasion abilities of POU4F2‐knockdown HCT116 (C) and POU4F2‐knockdown RKO (D) cells. E, F , Expression of E‐cadherin and vimentin proteins in POU4F2‐knockdown HCT116 (E) and POU4F2‐knockdown RKO (F) cells. G, H, Expression of EMT‐TFs in POU4F2‐knockdown HCT116 (G) and POU4F2‐knockdown RKO (H) cells. All experiments were performed in triplicate. Data were presented as the mean ± SEM (*P < .05, **P < .01, ***P < .001, ****P < .0001)